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In these problems, it has been shown that the rise of endogenous Palmitoylethanolamide—both by lowering its degradation or exogenous administration—can preserve neuroinflammation within its physiological restrictions. On this evaluation the large variety of scientific tests on the advantages derived from oral administration of micronized and re
PEA protects against bacterial infection by means of innate immune modulation involving MCs, macrophages and microglia. Prophylactic PEA at a dose of 0.one mg/kg overall body weight prolonged survival charge and minimized neuro-inflammation in an aged murine bacterial meningitis product, in the absence of antibiotics [42].Injuries can be a part of
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